TEST · C19H28O2
A:A → 100:100
AROMATIZES
5α-REDUCES
REFERENCE
Endogenous AR agonist — the gold-standard baseline. CYP19A1→E2 aromatization. 5α-reductase→DHT conversion. All ester half-lives: Suspension <1d / Prop ~2d / E ~7d / C ~8d.
TEST_ENANTHATE_250$280
TEST_CYPIONATE_250$280
TEST_PROPIONATE_100$240
TEST_SUSPENSION_100$230
NANDROLONE · DECA / NPP
A:A → 125:37
5α→DHN INACTIVE
PR AGONIST
19-nortestosterone. C-19 methyl group removed. 5α-reduction yields DHN (inactive) → selectively low androgenicity. ~20% PR activity. DECA: 15-day t½. NPP: 5-day t½.
DECA_200MG_10VL$310
NPP_100MG_10VL$280
TRENBOLONE · ACE / ENAN
A:A → 500:500
NO AROMATIZE
Δ9,11 STRUCTURE
3–5× AR AFFINITY
19-nor triene with Δ4,9,11 double bonds. Δ9 blocks CYP19A1 (no aromatization). Δ11 blocks 5α-reductase. AR affinity 3–5× testosterone. Strong PR (~60% progesterone). 500:500 uniform potency = 5× baseline.
TREN_ACE_100MG_10VL$320
TREN_ENAN_200MG_10VL$350
DIANABOL · DBol
A:A → 210:60
17α-METHYL
17α-ME-E2
ORAL BIO
17α-methyl Δ1-testosterone. C17α methylation blocks hepatic first-pass → oral bioavailability. Aromatizes to 17α-methylestradiol (more potent than E2, AI-resistant). Also available as injectable oil for first-pass comparison research.
DBOL_10MG_100CT$140
DBOL_50MG_100CT$160
DBOL_INJ_50MG_ML$310
DBOL_50MG_1000CT_BULK$600
ANAVAR · OXANDROLONE
A:A → 322:24
2-OXA RING
NO AROMATIZE
RENAL CLEARANCE
DHT + 2-oxa A-ring substitution. Highest anabolic selectivity of any common oral AAS (322:24 = 13.4× selectivity ratio). Cannot aromatize or 5α-reduce. Predominantly renal excretion → lower hepatotoxicity vs other 17α-AA.
WINSTROL · STANOZOLOL
A:A → 320:30
PYRAZOLE A-RING
SHBG DISPLACER
BOTH 17α-AA
DHT + pyrazole A-ring — unique among all AAS. Both oral AND injectable forms are 17α-alkylated (injectable is aqueous suspension, ~24h from depot, not ester-based). Highest SHBG affinity → frees other steroids. 320:30 ratio.
WINNY_10MG_100CT$130
WINNY_50MG_100CT$160
WINNY_INJ_50MG_ML$270
MASTERON · DROSTANOLONE
A:A → 62:25
2α-METHYL DHT
CYP19A1 INHIBITOR
REDUCES E2
2α-methyl DHT. Directly inhibits aromatase (CYP19A1) — the only AAS that simultaneously acts as an AR agonist AND aromatase inhibitor. Steroidal AI. Masteron Prop: ~2-day t½. Masteron Enan: ~7-day t½.
MAST_PROP_100MG_10VL$290
MAST_ENAN_200MG_10VL$320
ANADROL · OXYMETHOLONE
A:A → 320:45
DIRECT ER AGONIST
AI-RESISTANT
2-OHME GROUP
DHT + 2-hydroxymethylene group. 320:45 ratio. Does NOT aromatize — but directly activates estrogen receptor (ER) as an AI-resistant ER agonist. This makes it unique: E-side effects cannot be controlled by aromatase inhibitors.
ANADROL_50MG_100CT$150
ANADROL_INJ_50MG_ML$330
MENT · TRESTOLONE ACE
A:A → 650:250
7α-METHYL
HIGHEST RATIO
MALE CONTRACEPTIVE
7α-methyl-19-nortestosterone acetate. 650:250 — highest A:A of any injectable AAS. 5α→7α-Me-DHN (active — maintains androgenicity unlike Nandrolone). Aromatizes to 7α-methylestradiol. Strong PR. Male contraceptive research lead.
EQUIPOISE · BOLDENONE UU
A:A → 100:50
LONGEST t½
EPO STIMULATION
50% AROMATIZE
Δ1-testosterone + C11 undecylenate ester. ~14-day half-life — longest of any common injectable AAS. 50% testosterone aromatization rate. Stimulates EPO production exceeding testosterone. 300mg/mL high-concentration vials.